World Journal of
Pharmaceutical and Life Sciences

THE SPIKE PROTEIN INFECTION IMPACT ON HOMO SAPIEN SKIN BLACK IS THE LEAST WHEREAS UPON HOMO SAPIEN SKIN WHITE IS THE MOST

Dr. S. S. Sawhney*

ABSTRACT

Homo sapien skin black race had been the only race, defined and conceptualized at tropics with the involvement of high intensity UV rays with life’s basic facilities demand and pressure. The original Homo sapien skin black race had to migrate to non-tropics, defined with very lowlevel UV rays with no ability of photoreact with vitamin K epidermally placed at threshold level, causing Homo sapiens to turn skin white at non-tropics. It is environmental effects upon earth only. The layering of melanolipoprotein over stratum granulosum causes saturation in catenation or hydrogen bonding with no space at stratum granulosum for any other addition to step in, whereas the Homo sapien skin white race sans melanolipoprotein gives free access to viruses, bacteria and pathogens etc to hydrogen bond to the exposed protein driven white skin. The spike protein with defined –C=O and –OH groups have the tendency to hydrogenbond with complementary –OH and –C=O functional groups of exposed protein of skin white race whereas the Homo sapien skin black race does not allow functional groups of the spike protein to catenate the black skin as the stratum granulosum is fully catenated and saturated with no further bonding. The spike protein can not infect Homo sapien black skin whereas there are 100% chances for spike protein toinfect 100% the white skin.

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